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Calcitriol restrains microglial M1 polarization and alleviates dopaminergic degeneration in hemiparkinsonian mice by boosting regulatory T-cell expansion

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成果类型:
期刊论文
作者:
Xie, Yangzhi;Chen, Liang;Chen, Jiacheng;Chen, Yongjun
通讯作者:
Chen, YJ
作者机构:
[Chen, Liang; Chen, Yongjun; Xie, Yangzhi] Univ South China, Affiliated Nanhua Hosp, Hengyang Med Sch, Dept Neurol, Hengyang 421001, Peoples R China.
[Chen, Jiacheng] Univ South China, Affiliated Nanhua Hosp, Hengyang Med Sch, Dept Intens Care Unit, Hengyang, Peoples R China.
通讯机构:
[Chen, YJ ] U
Univ South China, Affiliated Nanhua Hosp, Hengyang Med Sch, Dept Neurol, Hengyang 421001, Peoples R China.
语种:
英文
关键词:
M1/M2 polarization;neuroinflammation;Parkinson's disease;regulatory T cell;vitamin D receptor
期刊:
Brain and Behavior
ISSN:
2162-3279
年:
2024
卷:
14
期:
2
页码:
e3373-
基金类别:
Key Project of Hunan Provincial Health and Health Commission in 2020
机构署名:
本校为第一且通讯机构
院系归属:
医学院
摘要:
We explored the therapeutic effects of vitamin receptor activation by calcitriol in a mouse model of PD. We found that calcitriol is potent to protect against neuroinflammation and neuronal degeneration by boosting Treg. Vitamin D receptor may be a therapeutic option for PD. Abstract Objective Vitamin D deficiency is a risk factor for Parkinson's disease (PD) and vitamin D supplementation robustly alleviates neurodegeneration in PD models. However, the mechanisms underlying this effect require further clarification. Current evidence suggests th...

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