Overexpression of miR-3196 suppresses cell proliferation and induces cell apoptosis through targeting ERBB3 in breast cancer

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成果类型：

期刊论文

作者：

Ji, Z-C;Han, S-H;Xing, Y-F*

通讯作者：

Xing, Y-F

作者机构：

[Ji, Z-C] Changji Huizu Peoples Hosp Xinjiang, Dept Gen Surg, Xinjiang, Peoples R China.

[Han, S-H] Univ South China, Affiliated Hosp 1, Dept Med Oncol, Hengyang, Peoples R China.

[Xing, Y-F] Tongchuan Min Bur, Cent Hosp, Dept Gen Surg, Tongchuan, Peoples R China.

通讯机构：

[Xing, Y-F] T

Tongchuan Min Bur, Cent Hosp, Dept Gen Surg, Tongchuan, Peoples R China.

语种：

英文

关键词：

Apoptosis;Breast cancer;ERBB3;MiR-3196;Proliferation

期刊：

EUROPEAN REVIEW FOR MEDICAL AND PHARMACOLOGICAL SCIENCES

ISSN：

1128-3602

年：

2018

卷：

期：

页码：

8383-8390

DOI：

10.26355/eurrev_201812_16536

机构署名：

本校为其他机构

院系归属：

医学院

摘要：

OBJECTIVE: Breast cancer is one of the most common malignancies worldwide. MicroRNAs (MiRNAs) have been identified to influence cell behaviors through epigenetic post-transcriptional gene regulation. Therefore, the aim of this study was to determine the role of miR-3196 in the proliferation and apoptosis of breast cancer. MATERIALS AND METHODS: Human breast cancer cell lines (MCF-7 and MDA-MB-231) were obtained and cultured. The expression level of miR-3196 in breast cancer tissues was detected using Real Time-Polymerase Chain Reaction (RT-PCR)...

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Overexpression of miR-3196 suppresses cell proliferation and induces cell apoptosis through targeting ERBB3 in breast cancer

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